Skip to main content
Fig. 6 | Genome Biology

Fig. 6

From: Chromatin spatial organization of wild type and mutant peanuts reveals high-resolution genomic architecture and interaction alterations

Fig. 6

Assay for Transposase-Accessible Chromatin using sequencing (ATAC-seq) analysis in peanut. A Normalized read signal of ATAC-seq peak sites in H2014, the wild type (left) and H1314, the dwarf mutant (right). Numbers in the bottom box show stronger (red) and weaker (green) ATAC-seq signals in H1314, respectively. B The motif sequences identified around the stronger ATAC-seq peaks in H1314, including MYB (top), ESE1 (AP2EREBP) (center left), AT5G23930 (mTERF) (center right), ERF4 (AP2EREBP) (bottom left), and ERF10 (AP2EREBP) (bottom right). C Genomic location distribution of increased accessible chromatin signals in H1314. The ratio shows the ascending arrangement as distal intergenic (89.56%), promoter-TSS (4.01%), exon (3.00%), TTS (2.58%), and intron (0.86%). D KEGG pathway of the nearest gene located in genomic regions with stronger ATAC-seq signals in H1314. E GO enrichment of the nearest gene located in genomic regions with stronger ATAC-seq signals in H1314. Genes in D and E were identified as the putatively regulated target genes based on that the ATAC-seq peaks were assigned to its nearest transcription start site

Back to article page